Grants

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C-Trap Optical Tweezers System for Single-Molecule Studies

OD - NIH Office of the Director

PROJECT SUMMARY / ABSTRACT The goal of this Shared Instrumentation Grant application is to permanently integrate a LUMICKS C-Trap Dymo optical tweezers system into the Structural Biology Core (SBC) at UT Health San Antonio (UTHSA), fulfilling a critical gap in our ability to visualize and manipulate biomolecular interactions in real time. UTHSA’s SBC has long provided regional leadership in structural biology, supporting NIH-funded research through cryo- EM, X-ray crystallography, and NMR. However, over a dozen NIH-funded investigators on campus study intrinsically disordered proteins (IDPs), phase-separated condensates, and force-sensitive macromolecular complexes – systems that cannot be fully resolved using traditional methods. The C-Trap uniquely addresses this limitation by enabling single-molecule interrogation of full-length, dynamic protein and nucleic acid assemblies as they respond to protein binding partners, nucleic acid scaffolds, small molecule ligands, and mechanical forces, all in real time. Already deployed on campus under a temporary lease agreement funded through a collaborative, grassroots effort by three NIH-funded investigators, the C-Trap has catalyzed transformative discoveries in just three months of operation. This model, while unsustainable, has demonstrated the platform’s immediate value and feasibility at UTHSA, enabling high-quality, novel data collection in genome maintenance, transcriptional regulation, and cancer biology. As the first and only C-Trap Dymo system in Texas, its presence has already begun to transform research approaches across our institution. Formal integration into the open-access SBC is urgently needed to expand availability, ensure operational continuity, and unlock its full potential for a growing community of NIH-funded structural, cancer, and cellular researchers. This proposal now seeks to broaden access by embedding the C-Trap within the SBC, where it will enhance training capacity, sustainability, and resource sharing across UTHSA, the University of Texas System, and regional collaborators. It will also complement cryo-EM, NMR, and X-ray capabilities already housed in the SBC, solidifying UTHSA’s emerging leadership in structural biology and single-molecule biophysics. If funded, the S10 will provide permanent institutional access to the already-installed instrument and support a dedicated staff scientist for operations and user training. With investment in this high-value, high-impact technology, the C-Trap will continue to accelerate scientific discovery, enable cutting-edge experimentation, strengthen the research portfolios of new and established investigators, and expand the state of Texas’s capacity for frontier biomedical research.

Up to $1.0M

2027-06-30

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C. albicans Induced Immune Skewing in Early Life

NIAID - National Institute of Allergy and Infectious Diseases

PROJECT ABSTRACT There is a growing epidemic of immune-mediated diseases affecting pediatric populations. Candida albicans is an opportunistic fungal species that is habitually found as part of the microbiota in humans, particularly as part of a “dysbiotic” perturbed microbiota associated with increased immune-mediate inflammation disease. Clinical observations have found a strong association with C. albicans and inflammatory diseases, though it is unclear if C. albicans initiates inflammation in an already at-risk atopic individual through acute infection, or can directly influence the development of immune responses. C. Albicans can reside within the intestinal microbiota, and exposure to C. albicans in both human and mice results in the anti-C. albicans immunoglobulins, suggesting an antigen specific immune response. This project will ask how intestinal exposure to C. albicans in early life influences the developing immune system, specifically the T cell responses. Fungal species, including C. albicans induce T cell responses that produce the inflammatory cytokine IL-17, also referred to as T helper 17 cells (Th17), and presence of fungal species can induce further microbial dysbiosis within the intestinal microbiota. It is unknown whether the Th17 cells are responding to the C. albicans or dysbiotic bacterial species in the perturbed microbiota. Additionally, animal studies of C. albicans are hampered by a decreased ability to colonize the adult murine intestinal microbiota with C. albicans without antibiotic perturbation. We have developed a model of neonatal C. albicans acquisition where oral administration of C. albicans between postnatal day 7 and 10 results in stable, robust C. albicans colonization, which is associated with microbial dysbiosis, increase in systemic IL17, increase in Th17 cells in the intestine, and disrupted regulatory T cells. To expand upon these findings, we will 1) evaluate the requirement of goblet cell-associated antigen passages in the induction of a lasting IL-17 response during early life, to assess how intestinal antigens inducing IL-17 responses are introduced to the intestinal immune system following C. albicans colonization. Next, we will 2) evaluate the ability of C. albicans to induce specific Th17 cells and increase pathology during psoriasis using tetramers for C. albicans specific cells, and a chemical-contact model of dermatitis. Through these aims we will mechanistically connect preliminary data from our novel animal model to immune perturbations following C. albicans colonization. Following the completion of this project, we will understand the nature of the antigens initiating Th17 cell responses and have a robust platform to assess the consequence of increased Th17 cells and inflammatory responses following C. albicans colonization. This work has important implications in understanding dysbiosis and microbial imprinting of the immune system in early life.

Up to $444K

2028-01-31

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C. auris mannosylation genes impact neutrophil evasion

NIAID - National Institute of Allergy and Infectious Diseases

PROJECT SUMMARY Since the identification of the fungal pathogen clinically known as Candida auris in 2009, the incidence of infections caused by C. auris has increased around the world. (This pathogen has recently been reclassified by phylogenomic analysis as Candidozyma auris. Throughout this proposal, I refer to this pathogen as C. auris or Candida auris, the name used clinically.) In fact, between 2019 and 2022, the incidence of clinical C. auris infections reported in the United States increased five-fold. C. auris spreads rapidly within healthcare facilities, particularly critical care units. In addition, C. auris is often drug resistant. Nonetheless, with targeted antifungal therapy, invasive C. auris infection has a crude mortality rate of about 35%. Neutrophils are a crucial defense against many invasive fungal infections, including candidiasis. However, primary human neutrophils exhibit minimal activity against Candida auris in comparison to Candida albicans. There is a critical need to understand immune evasion and, particularly, neutrophil evasion mechanisms for this fungal pathogen with the goal of developing new treatment modalities for invasive infection. This project is focused on identifying neutrophil evasion properties of the C. auris mannans, the polysaccharide chains making up the outermost component of the cell wall. C. auris mannans have been shown to mask β- glucan and chitin, known immunostimulatory motifs in the fungal cell wall. Our lab is interested in defining evasion mechanisms of mannans beyond this masking. We have identified C. auris mannan mutants that are more likely to be engulfed by human neutrophils but do not have increased β-glucan or chitin exposure on the fungal cell surface. I will use these mannosylation mutants of C. auris to define how specific genes impact neutrophil receptor recognition of this pathogen. I will also generate fungal-like mannan-coated particles by binding isolated mannans to functionalized beads and examine mannan-receptor interactions independent of other cell wall components. In addition, I will develop a zebrafish model of invasive C. auris infection following skin disruption. This model will mimic the route of infection seen in hospital settings and allow for in vivo imaging of fungal-neutrophil interactions. The rich academic environment at the University of Wisconsin-Madison offers the medical, scientific, and graduate training resources necessary to actualize the proposed work. The proposal combines the fungal and neutrophil biology expertise of the sponsor and co-sponsor as well as their commitment to training the next generation of physician scientists. Overall, this predoctoral fellowship provides training to achieve the research, clinical, mentorship, leadership, and communication skills necessary to develop as an independent physician scientist investigator.

Up to $46K

2029-05-31

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C1DA--A/E Replace Existing Substation A9 Project 642-26-117 NCO4 Construction East

VETERANS AFFAIRS, DEPARTMENT OF.VETERANS AFFAIRS, DEPARTMENT OF.244-NETWORK CONTRACT OFFICE 4 (36C244)

C1DA--A/E Replace Existing Substation A9 Project 642-26-117 NCO4 Construction East

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C1DZ--621-24-701 EHRM Infrastructure Upgrades Tier 3 Data Center Construction - Mountain Home VAMC

VETERANS AFFAIRS, DEPARTMENT OF.VETERANS AFFAIRS, DEPARTMENT OF.PCAC (36C776)

C1DZ--621-24-701 EHRM Infrastructure Upgrades Tier 3 Data Center Construction - Mountain Home VAMC

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C2 Cross Functional Team (CFT) Broad Agency Announcement

DEPT OF DEFENSE.DEPT OF THE ARMY.AMC.ACC.ACC-CTRS.ACC-APG.W6QK ACC-APG

C2 Cross Functional Team (CFT) Broad Agency Announcement

2030-03-23

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C5ISR and other space-related products

European Commission - EDF

C5ISR and other space-related products. Call: 2nd call for proposals for common procurement actions under the Programme. Programme: EDF. Open call from the EU Funding & Tenders Portal.

$50K

2027-02-16

technology

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CA Film Commission Tax Credit & Grants

California Film Commission

Incentives and grants for film, TV, and digital media production in California.

$5K – $100K

filmtourismarts

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CABOT TOWN OF BETTER ROADS

Transportation Agency

CABOT TOWN OF BETTER ROADS

Up to $7K

2027-10-31

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Cadmium-induced Pulmonary Arterial Hypertension Pathogenesis and Underlying Mechanisms

NIEHS - National Institute of Environmental Health Sciences

Prevalence of pulmonary arterial hypertension (PAH) is increasing worldwide with five-year survival <50%. There is no effective therapy to prevent right ventricular (RV) failure in PAH, urging the need to investigate its pathogenic mechanisms to develop novel preventive and therapeutic approaches. Experimental models and clinical data showed that oxidative stress appears as a significant mediator and predictor of worse outcomes in PAH. Noteworthy, exposures to nonessential metals, such as cadmium (Cd), have been linked to increased incidence of cardiovascular diseases. However, no study has investigated the association of chronic environmental exposure to Cd with PAH although some indirect findings available. Our preliminary metallomics data showed, when compared to controls, both lung and cardiac Cd levels significantly increased in male mice received 5 ppm Cd exposure in the drinking water for 12 weeks (wks). Pulmonary artery collagen accumulation and muscularization severity as one of the key pathogeneses in PAH worsened in the lung of mice exposed to 12-wks Cd, a PAH-like pathogenesis of mice with PAH induced by SuHx a well characterized rodent PAH model. Metallomics analysis revealed significant Fe accumulations in the lungs of Cd-exposed and SuHx-induced PAH mice. However, whether longer Cd exposure can induce or exacerbate PAH pathogenesis, and whether these can be replicated in female mice remain unknown. We thus hypothesize that chronic exposure to Cd at environmental levels for 16-wks can induce PAH or exacerbate SuHx-induced PAH pathogenesis, which is because Cd may cause iron dyshomeostasis and associated ferroptosis. Aim 1: To comprehensively define whether chronic Cd exposure can directly induce, and/or exacerbate PAH pathogenesis and essential metal homeostasis in the heart and lungs. We will have three groups, 1) Cd exposure model for 16 wks in both male and female mice; 2) Pre-PAH Cd model will be given Cd in the drinking water for 16 weeks meanwhile PAH will be induced with Su5416/hypoxia (SuHx) during the last 4 wks; 3) Post-PAH Cd model will be similar to group #2, except for PAH induction during the first 4 wks. For all 3 models, cardiac functions will be examined with echocardiography before and after PAH induction and every 4 weeks until the end of the 16-wk exposure. Mice will be euthanized at the end of 16 weeks to collect blood, lungs, and heart for PAH pathogenesis analysis. Aim 2: To define the mechanism of Cd-induced Fe redistribution and Fe-mediated ferroptosis in PAH pathogenesis. Aim 2a will analyze Cd and essential metals in the blood and tissues by metallomics, Fe metabolism related transporters and mediators, and lung and cardiac ferroptosis. Aim 2b will apply laser capture collection of pulmonary arteries from the lungs of mice with and without Cd collected in Aim 1, for proteomic analysis determining distinct protein expression profiles to reveal novel insights into mechanisms for Cd-caused Fe redistributions, ferroptosis, and signaling pathways. Aim 2c will define pathogenic roles of ferroptosis in PAH pathogenesis and RV dysfunction in vivo using ferroptosis inhibitor and Fe chelator in mouse models.

Up to $426K

2028-04-22

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Cady Brook Bridge & Five Mile River Bridge

Putnam

Cady Brook Bridge & Five Mile River Bridge

Up to $594K

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CAGE-Enabled Single-Lineage RNA-Seq for Bacterial PERTURB-Seq and Host-Pathogen Interaction Analysis

NIAID - National Institute of Allergy and Infectious Diseases

Project Summary This R21 pilot grant aims to benchmark single-lineage RNA-Seq (slRNA-Seq), an innovative technology that enables transcriptome analysis of microbial lineages with sensitivity far exceeding current single-microbe methods. Current approaches for bacterial RNA-seq are severely limited by technical challenges including short mRNA half-lives, absence of poly-A tails, and small transcript numbers. We established an approach using “Capsules with Amphiphilic Gel Envelopes” (CAGEs) that allows clonal expansion of individual cells followed by multi-step in-capsule library preparation, dramatically improving transcript detection to thousands of genes per lineage while efficiently capturing CRISPR interference perturbations. Building on this, this pilot grant will evaluate the broad utility of slRNA-Seq through two specific aims: (1) evaluate bacterial PERTURB-Seq by optimizing library preparation protocols and generating a benchmark dataset of all transcription factor perturbations in E. coli; and (2) develop slRNA-Seq as a tool for host- pathogen interaction profiling through co-encapsulation of human immune cells with microbial populations. Our specific focus will be on detecting immune cell transcriptional responses alongside pathogen identity in each CAGE and evaluating that CAGEs are not perturbative to immune cells or their response. We propose multiple strategies to optimize CAGEs and slRNA-Seq if we identify challenges during the pilot. This technology directly addresses NIAID priorities for tools to systematically dissect host-pathogen responses. If successful, this project will establish slRNA-Seq as a robust platform for bacterial functional genomics and microbiome research, providing the scientific community with transformative tools to investigate previously intractable questions about microbial gene function and host-pathogen interactions.

Up to $296K

2028-04-30

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Cahtolic Charities

Cahtolic Charities

Single-Family Detached

Up to $3K

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Cahtolic Charities

Cahtolic Charities

Mobile Home

Up to $9K

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Cal Anderson Park Communication Hub

Cal Anderson Park Emergency Communications Hub

The Cal Anderson Park Emergency Communication Hub will be hosting two community events, the first on August 12th the "Hub Grand Opening" and the second will take place September 7th "The Urban Survival Skills Fair". Both events will take place at Cal Anderson Park at the Sun Bowl/Patio located at 1635 11th Ave W. The purpose of both events is to empower the Capitol Hill neighborhood with the knowledge and skills in preparedness and disaster training. Relationships and partnerships will be made and community is encourage to participate in the variety of skills trainings at the fair. The event is free and open to the public.

Up to $5K

Education

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CAL FIRE Forest Health Research Program (FY 2026-27) California Climate Investments Graduate Student Grants (RP-RFP-2026-02)

environment & water; sciencetechnologyand research & development

Department of Forestry and Fire Protection

The application will consist of a concept proposal followed by review and selection. Selected applicants will be invited to submit a full proposal followed by a second review and selection period. Concept proposals are due by 3 p.m. (PDT), July 30, 2026. Full invited proposals are due by 3 p.m. (PST), November 18, 2026. The Forest Health Research Program (hereafter “Research Program”) was established as part of CAL FIRE’s plan for implementing the California Forest Carbon Plan. It is one of several CAL FIRE programs funded through the California Climate Investments (CCI) program, Greenhouse Gas Reduction Fund. The mission of the Research Program is: To identify and prioritize research topics in forest health and fire science critical to the State of California To fund sound scientific studies that support forest landowners, resource agencies, and fire management organizations within the state To ensure scientific information generated from the program is made available to support decision making and policy To further the goals of the Wildfire and Forest Resilience Action Plan, California Forest Carbon Plan, the California Natural and Working Lands Implementation Plan, CCI, and AB 32 Global Warming Solutions Act The program, procedures and requirements apply only to “stand-alone research” projects, where research-related activities are the only activities proposed. The following topics have been identified as priorities for study for the Research Program for FY 2026-27: Recovery, restoration, regeneration and reforestation strategies following wildfires and other disturbances in an altered future climate. Implementation, effectiveness, impacts, and tradeoffs of current and alternative management strategies to reduce unwanted wildfire impacts to communities, improve hydrologic functions including water quality and supply, improve air quality, sustain and promote biodiversity, mitigate impacts of pests, pathogens, and invasive species, and/or increase beneficial fire effects. Methods of ecological monitoring to continually evaluate effectiveness, impacts and benefits of vegetation treatments at various scales. Community resilience to wildfires and other ecological disturbances under current and future climate conditions. Traditional ecological knowledge (TEK) and tribal forest and fire management, especially research by tribal organizations or indigenous researchers. Forest products and utilization of forest residues related to fuel reduction and forest health treatments.Improved prediction of wildland fire spread, behavior, severity, and potential impacts, particularly under extreme weather conditions and/or within the wildland-urban interface. In addition, the following special topic has been identified as priorities for the Research Program for FY 2026-27: Utilizing novel applications of FireSat Quick Beam Instrument data to answer ecological questions.

Up to $500K

2026-07-30

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CAL FIRE Forest Health Research Program (FY 2026-27) California Climate Investments Grants (RP-RFP-2026-01)

environment & water; sciencetechnologyand research & development

Department of Forestry and Fire Protection

The application will consist of a concept proposal followed by review and selection. Selected applicants will be invited to submit a full proposal followed by a second review and selection period. Concept proposals are due by 3 p.m. (PDT), July 30, 2026. Full invited proposals are due by 3 p.m. (PST), November 18, 2026. The Forest Health Research Program (hereafter “Research Program”) was established as part of CAL FIRE’s plan for implementing the California Forest Carbon Plan. It is one of several CAL FIRE programs funded through the California Climate Investments (CCI) program, Greenhouse Gas Reduction Fund. The mission of the Research Program is: To identify and prioritize research topics in forest health and fire science critical to the State of California To fund sound scientific studies that support forest landowners, resource agencies, and fire management organizations within the state To ensure scientific information generated from the program is made available to support decision making and policy To further the goals of the Wildfire and Forest Resilience Action Plan, California Forest Carbon Plan, the California Natural and Working Lands Implementation Plan, CCI, and AB 32 Global Warming Solutions Act The program, procedures and requirements apply only to “stand-alone research” projects, where research-related activities are the only activities proposed. Research activities are no longer eligible as a component of larger management-oriented Forest Health projects. The following topics have been identified as priorities for study for the Research Program for FY 2024-25. Topics are numbered for reference, but not ranked in terms of priority. Research proposed under this solicitation must address one or more of these priority topics. Research projects should be focused on and relevant to California ecosystems and their management. Recovery, restoration, regeneration and reforestation strategies following wildfires and other disturbances in an altered future climate. Implementation, effectiveness, impacts, and tradeoffs of current and alternative management strategies to reduce unwanted wildfire impacts to communities, improve hydrologic functions including water quality and supply, improve air quality, sustain and promote biodiversity, mitigate impacts of pests, pathogens, and invasive species. Methods of ecological monitoring to continually evaluate effectiveness, impacts and benefits of vegetation treatments at various scales. Community resilience to wildfires and other ecological disturbances under current and future climate conditions. Traditional ecological knowledge (TEK) and tribal forest and fire management, especially research by tribal organizations or indigenous researchers. Forest products and utilization of forest residues related to fuel reduction and forest health treatments. Improved prediction of wildland fire spread, behavior, severity, and potential impacts, particularly under extreme weather conditions and/or within the wildland-urban interface. Improved methods and applications for conducting cost-benefit analysis related to forest restoration, fuel treatments and other forest health interventions.

Up to $4M

2026-07-30

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CAL FIRE Forest Health Research Program (FY 2026-27) Proposition 4 California Climate Bond Grants (RP-RFP-2026-03)

environment & water; sciencetechnologyand research & development

Department of Forestry and Fire Protection

The application consists of a concept proposal followed by review and selection. Selected applicants will be invited to submit a full proposal followed by a second review and selection period. Concept proposals are due by 3 p.m. (PDT), July 30, 2026. Full invited proposals are due by 3 p.m. (PST), November 14, 2026. This solicitation funds collaborative research to support forest health and greenhouse‑gas reduction at landscape scales through improved forest and vegetation management across California. This solicitation is purposefully intended to consider research proposals that focus on a broad range of challenging questions relevant to forest and fire management at large spatial scales, and we encourage applications from any discipline that meaningfully intersect with landscape-scale management. Research funded through this solicitation should be collaborative in nature, and include multiple partners working across organizations, institutions, jurisdictions, and/or disciplines. Projects should substantially advance their field of research and produce meaningful applied benefits for any of the following broad themes: a) improved forest or vegetation management strategies and activities to reduce unwanted disturbance impacts, promote recovery after disturbance, enhance carbon storage, sustain and promote biodiversity, improve water and air quality, provide regional economic benefits, or facilitate an adaptive management feedback loop (including beneficial fire, tribal stewardship, forest fuels reduction, pest management, reforestation, biomass utilization, forest watershed restoration, upper watershed, riparian, and mountain meadow restoration) at landscape-scales; b) Improved decision support for planning and prioritizing risk reduction activities, including large-scale prescribed fire, across scales from Potential Operational Delineations (PODs) to individual projects. Improvements may include but are not limited to fire behavior modeling, fuels quantification, andprioritization schema. c) Improved understanding of current impacts of large-scale wildfires and other large disturbances, or management strategies within large disturbance footprints, such as second-entry treatments in fire footprints; d) Improved predictions of future conditions, disturbance regimes, or treatment effectiveness; e) Emissions reductions and/or avoided live vegetation losses, improved long-term carbon storage and sequestration, or improved quantitative assessment of greenhouse gas impacts across large scales; Or f) Improved policy related to the California Forest Carbon Plan or other State climate mitigation and adaptation strategies. The research may – but is not required to – include forest treatment implementation such as forest fuels reduction, pest management, reforestation, biomass utilization, forest watershed restoration, upper watershed, riparian, and mountain meadow restoration. The research may – but is not required to – build off other previous or current implementation or research projects funded through other sources (e.g. Forest Health, Fire Prevention, Tribal Wildfire Resiliency, or other CAL FIRE or non-CAL FIRE grants).

$1M – $1.5M

2026-07-30

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Cal Grant Program

financial aidscholarshipshigher education

California Student Aid Commission

State scholarship and financial aid program administered by California Student Aid Commission for CA students and educational institutions.

$1K – $15K

2027-03-02

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CALAIS TOWN OF ER E23-6(623) E23-6(624)

Transportation Agency

CALAIS TOWN OF ER E23-6(623) E23-6(624)

Up to $70K

2027-04-30

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CALAIS TOWN OF ER E24-3(664)

Transportation Agency

CALAIS TOWN OF ER E24-3(664)

Up to $5K

2027-05-30

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Calcified Neurocysticercosis: New Insights Into Brain inflammation, Damage and Epileptogenesis

NINDS - National Institute of Neurological Disorders and Stroke

Abstract: Neurocysticercosis (NCC) is the most common helminthic infection of the human central nervous system caused by the larval stage of the pork tapeworm, Taenia solium. Calcification is a common outcome in NCC that carries increased risk for seizures, hippocampal atrophy and sclerosis. Approximately 30% of epilepsy cases in endemic regions are attributed to NCC. The disease is increasingly seen in non-endemic regions due to increased migration worldwide. Calcified granulomas are the most common radiologic finding and are responsible for the burden of disease, yet little research is focused on this important manifestation of NCC. This project aims to advance fundamental knowledge about calcified NCC; the pathophysiological mechanisms leading to calcification are basically unknown. This meeting will focus on establishing collaborative research networks to prioritize the study of calcified NCC, enhance diagnostic and therapeutic strategies, and ultimately improve health outcomes for affected populations by reducing seizure risk and brain inflammation. It will have broad implications for understanding epileptogenesis, neuroinflammation, and scarring. This meeting also includes early-stage investigators and different perspectives on NCC and will reach a large body of researchers in the field by recording and distributing the sessions. Ultimately, this project will amplify the topic of calcified NCC, which contributes to ongoing morbidity worldwide yet is rarely addressed in research questions, because it would be the first meeting of its kind to focus on this topic.

Up to $15K

2027-04-30